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By Anderson, Ian; Honkala, Iiro

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147, 41–61. J. (2003). Studies on BrdU labeling of hematopoietic cells: stem cells and cell lines. J. Cell. Physiol. 197, 251–260. Pantel, K. H. (2004). Dissecting the metastatic cascade. Nat. Rev. Cancer 4, 448–456. H. and Xiong, Y. (2005). Biochemical and cellular mechanisms of mammalian CDK inhibitors: a few unresolved issues. Oncogene 24, 2787–2795. H. (1998). Identification of a cyclin subunit required for the function of Drosophila P-TEFb. J Biol Chem. 273, 13855–13860. D. (2002). Not just a CDK inhibitor: regulation of transcription by p21(WAF1/CIP1/SDI1).

Ther. 2, S084–S095. M. (2004). Assays for cyclin-dependent kinase inhibitors. Methods Mol. Biol. 285, 69–78. M. (2005). Inhibitors of cyclin-dependent kinase modulators for cancer therapy, Prog Drug Res. 63, 183–206. S. (2002). A spindle checkpoint arrest and a cytokinesis failure by the dominant-negative polo-box domain of Plk1 in U-2 OS cells. J. Biol. Chem. 277, 32282–32293. , and Cittadini, A. (2003). Increased expression of cyclin E is associated with an increased resistance to doxorubicin in rat fibroblasts.

Fm Page 18 Saturday, September 2, 2006 12:09 PM 18 Inhibitors of Cyclin-dependent Kinases as Anti-tumor Agents attainment of induced and maintained dormancy as a positive therapeutic endpoint (Epstein, 2005). 5 MATHEMATICAL MODELS It is likely that intervention is never without unseen and unforeseen consequences. , 2004). , 2004). ” One implication is that the metrics for detection of response, usually favoring the more “active” cytotoxic induction modes of therapy, may be missing potentially important anticancer effects achieved by noncytotoxic maintenance modes of action (Epstein, 2005).

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